Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the other hand, our power to rescue SERT purpose in KI midbrain synaptoneurosomes through the inhibition of FAK signifies elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by improved pFAK localization in five-HT https://reubeny086boy8.bloginder.com/profile